prikaz prve stranice dokumenta Regulation of immune cell function and differentiation by the NKG2D receptor
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Scientific paper - Original scientific paper
Regulation of immune cell function and differentiation by the NKG2D receptor
Cellular and Molecular Life Sciences, 68 (2011), 21; 3519-3529. https://doi.org/10.1007/s00018-011-0797-0
Zafirova, Biljana; Wensveen, Felix M.; Gulin, Maja; Polić, Bojan

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Zafirova, B., Wensveen, F. M., Gulin, M. & Polić, B. (2011). Regulation of immune cell function and differentiation by the NKG2D receptor. Cellular and Molecular Life Sciences, 68. (21), 3519-3529. doi: 10.1007/s00018-011-0797-0

Zafirova, Biljana, et al. "Regulation of immune cell function and differentiation by the NKG2D receptor." Cellular and Molecular Life Sciences, vol. 68, no. 21, 2011, pp. 3519-3529. https://doi.org/10.1007/s00018-011-0797-0

Zafirova, Biljana, Felix M. Wensveen, Maja Gulin and Bojan Polić. "Regulation of immune cell function and differentiation by the NKG2D receptor." Cellular and Molecular Life Sciences 68, no. 21 (2011): 3519-3529. https://doi.org/10.1007/s00018-011-0797-0

Zafirova, B., et al. (2011) 'Regulation of immune cell function and differentiation by the NKG2D receptor', Cellular and Molecular Life Sciences, 68(21), pp. 3519-3529. doi: 10.1007/s00018-011-0797-0

Zafirova B, Wensveen FM, Gulin M, Polić B. Regulation of immune cell function and differentiation by the NKG2D receptor. Cellular and Molecular Life Sciences [Internet]. 2011 September 06 [cited 2024 April 21];68(21):3519-3529. doi: 10.1007/s00018-011-0797-0

B. Zafirova, F. M. Wensveen, M. Gulin and B. Polić, "Regulation of immune cell function and differentiation by the NKG2D receptor", Cellular and Molecular Life Sciences, vol. 68, no. 21, pp. 3519-3529, September 2011. [Online]. Available at: https://urn.nsk.hr/urn:nbn:hr:184:805924. [Accessed: 21 April 2024]

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Metadata
Title (english)Regulation of immune cell function and differentiation by the NKG2D receptor
AuthorBiljana Zafirova
AuthorFelix M. Wensveen
AuthorMaja Gulin
AuthorBojan Polić
Author's institutionUniversity of Rijeka
Faculty of Medicine
(Department of Histology and Embryology)
Scientific / art field,
discipline and subdiscipline		BIOMEDICINE AND HEALTHCARE
Basic Medical Sciences
Abstract (english)
NKG2D is one of the most intensively studied immune receptors of the past decade. Its unique binding and signaling properties, expression pattern, and functions have been attracting much interest within the field due to its potent antiviral and anti-tumor properties. As an activating receptor, NKG2D is expressed on cells of the innate and adaptive immune system. It recognizes stress-induced MHC class I-like ligands and acts as a molecular sensor for cells jeopardized by viral infections or DNA damage. Although the activating functions of NKG2D have been well documented, recent analysis of NKG2D-deficient mice suggests that this receptor may have a regulatory role during NK cell development. In this review, we will revisit known aspects of NKG2D functions and present new insights in the proposed influence of this molecule on hematopoietic differentiation.
Keywords (english)
Languageenglish
Publication typeScientific paper - Original scientific paper
Publication statusPublished
Peer reviewPeer review - international
Publication versionPublished version
Journal titleCellular and Molecular Life Sciences
Numberingvol. 68, no. 21, pp. 3519-3529
p-ISSN1420-682X
e-ISSN1420-9071
DOIhttps://doi.org/10.1007/s00018-011-0797-0
URN:NBNurn:nbn:hr:184:805924
Publication2011-09-06
Publication2011-11
ProjectNumber: 062-0621261-1271
Title (croatian): Uloga NKG2D u razvoju, homeostazi i efektorskim funkcijama imunološkog sustava
Leader: Bojan Polić
Jurisdiction: Croatia
Funding stream: ZP
ProjectNumber: 256686
Title (english): Upgrading the capacities for research in translational medicine at the Faculty of Medicine University of Rijeka
Acronym: TRANSMEDRI
Jurisdiction: eu
Funding stream: FP7
Document URLhttp://link.springer.com/10.1007/s00018-011-0797-0
Type of resourceText
Access conditionsOpen access
Terms of useLicence Attribution 4.0 International (CC BY 4.0) icon
Created on2018-11-30 07:32:46