Access restricted to higher education institution's students and staff
master's thesis
Endo - perio syndrome

Vika Kravanja (2012)
Metadata
TitlePulpoparodontni sindrom
AuthorVika Kravanja
Mentor(s)Ivana Brekalo-Pršo (thesis advisor)
Abstract
Pulpoparodontni sindrom je zajednički naziv za patološka stanja koja istovremeno zahvaćaju pulpu i parodont. Pulpa i parodont međusobno blisko komuniciraju zbog embrioloških, anatomskih i funkcionalnih veza (4). To je razlog što sva patološka stanja pulpe mogu neposredno i posredno utjecati na potporna tkiva zuba i obrnuto. U razvoju pulpoparodontnog sindroma sudjeluju različiti uzročnici koji mogu biti mikrobnog ili ne-mikrobnog podrijetla. U uzročnike mikrobnog podrijetla ubrajamo bakterije, viruse i gljive te njihove nusprodukte, dok su ne-mikrobni uzročnici neadekvatno endodontsko liječenje, koronarno propuštanje, traumatske ozljede, perforacije korijena te razvojne anomalije (7). Glavni oblici komunikacije između pulpe i parodonta jesu apikalni otvor, lateralni i akcesorni kanali i dentinski kanalići. U moguće oblike komunikacije svrstavaju se i furkacijski kanali, palatogingivalni, razvojni i apikalni žlijebovi te anahoreza (19). Najčešće rabljeni klasifikacijski sistem za podjelu pulpoparodontnih bolesti predložili su Simon, Glick i Frank 1972 godine. Klasifikacijski sistem obuhvaća pet različitih kategorija: 1. Primarno pulpna bolest 2. Primarno pulpna bolest sa sekundarno zahvaćenim parodontom 3. Primarno parodontna bolest 4. Primarno parodontna bolest sa sekundarno zahvaćenom pulpom 5. Prave kombinirane (pulpoparodontne) bolesti (23).
Keywordsclassification endo-perio syndrome etiologic factors infection interrelationship pulp periodontium route of communications.
Parallel title (English)Endo - perio syndrome
Committee MembersIvana Brekalo-Pršo
Snježana Glavičić Valovičić
Irena Glažar
GranterSveučilište u Rijeci
Medicinski fakultet
Lower level organizational unitsKatedra za endodonciju i restorativnu stomatologiju
PlaceRijeka
StateCroatia
Scientific field, discipline, subdisciplineBIOMEDICINE AND HEALTHCARE
Dental Medicine
Endodonics and Restorative Dentistry
Study programme typeuniversity
Study levelintegrated undergraduate and graduate
Study programmeDental Medicine
Academic title abbreviationdr. med. dent.
Genremaster's thesis
Language Croatian
Defense date2012-11-28
Parallel abstract (English)
Endo-perio syndrome is a common name for pathologic conditions that affect both the pulp and periodontium. The pulp and periodontium closely communicate through embryologic, anatomic and functional relationships (4). That is the reason that all pathologic conditions of the pulp can directly and indirectly affect the periodontium and vice versa . Many various factors participate in developing the endo-perio syndrome. These factors can either be of microbial or non-microbial origin. Microbial pathogens are bacteria, viruses and fungi and their by-products, while non-microbial causes are inadequate endodontic treatment, coronal leakage, traumatic injuries, root perforations and development malformations (7). The pulp and periodontium communicate through the apical foramen, lateral and accesory canals such as through dentinal tubules. Communication is also possible through furcation canals, palatogingival, development and apical grooves and anachoresis (19). The most commonly used classification system for the endo-perio syndrome was suggested by Simon, Glick and Frank in 1972. The classification system includes five different categories: 1. primary endodontic lesions 2. primary endodontic lesions with secondary periodontal involvement 3. primary periodontal lesions 4. primary periodontal lesions with secondary endodontic involvement 5. true combined lesions (23)
Parallel keywords (Croatian)pulpoparodontni sindrom pulpa parodont međuodnos infekcija etiološki čimbenici oblici komunikacije klasifikacija.
Resource typetext
Access conditionAccess restricted to higher education institution's students and staff
Terms of usehttp://rightsstatements.org/vocab/InC/1.0/
URN:NBNhttps://urn.nsk.hr/urn:nbn:hr:184:505201
CommitterBosa Licul