prikaz prve stranice dokumenta Luteolin ameliorates experimental colitis in  mice through ERK-mediated suppression of  inflammation, apoptosis and autophagy
No public access
Scientific paper - Original scientific paper
Luteolin ameliorates experimental colitis in mice through ERK-mediated suppression of inflammation, apoptosis and autophagy
Food and Chemical Toxicology, 145 (2020); 111680. https://doi.org/10.1016/j.fct.2020.111680


Cite this document

Vukelić, I., Detel, D., Batičić, L., Potočnjak, I. & Domitrović, R. (2020). Luteolin ameliorates experimental colitis in mice through ERK-mediated suppression of inflammation, apoptosis and autophagy. Food and Chemical Toxicology, 145.. doi: 10.1016/j.fct.2020.111680

Vukelić, Iva, et al. "Luteolin ameliorates experimental colitis in mice through ERK-mediated suppression of inflammation, apoptosis and autophagy." Food and Chemical Toxicology, vol. 145, 2020. https://doi.org/10.1016/j.fct.2020.111680

Vukelić, Iva, Dijana Detel, Lara Batičić, Iva Potočnjak and Robert Domitrović. "Luteolin ameliorates experimental colitis in mice through ERK-mediated suppression of inflammation, apoptosis and autophagy." Food and Chemical Toxicology 145 (2020). https://doi.org/10.1016/j.fct.2020.111680

Vukelić, I., et al. (2020) 'Luteolin ameliorates experimental colitis in mice through ERK-mediated suppression of inflammation, apoptosis and autophagy', Food and Chemical Toxicology, 145. doi: 10.1016/j.fct.2020.111680

Vukelić I, Detel D, Batičić L, Potočnjak I, Domitrović R. Luteolin ameliorates experimental colitis in mice through ERK-mediated suppression of inflammation, apoptosis and autophagy. Food and Chemical Toxicology [Internet]. 2020 [cited 2024 December 01];145. doi: 10.1016/j.fct.2020.111680

I. Vukelić, D. Detel, L. Batičić, I. Potočnjak and R. Domitrović, "Luteolin ameliorates experimental colitis in mice through ERK-mediated suppression of inflammation, apoptosis and autophagy", Food and Chemical Toxicology, vol. 145, 2020. [Online]. Available at: https://urn.nsk.hr/urn:nbn:hr:184:625064. [Accessed: 01 December 2024]

Please login to the repository to save this object to your list.